Projects
We investigate the pathophysiology of dry eye disease, with an emphasis on ocular surface inflammation, epithelial stress responses, and tissue homeostasis. In parallel, we develop and translate biomaterial-based therapeutic platforms designed to improve ocular surface protection and long-term treatment efficacy.
Featured
The Role and Molecular Mechanism of Bone Marrow-derived CXCR2-positive Monocytes in Hypertensive Retinal Vascular Injury.
Molecular Mechanism of Immunoproteasome Subunit β5i Promoting Angiotensin II-Induced Retinal Vascular Injury.
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